Reply to: “Correspondence to bipolar disorder-iPSC derived neural progenitor cells exhibit dysregulation of store-operated Ca2+ entry and accelerated differentiation” by Yde Ohki and colleagues
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Springer Science and Business Media LLC
Link
https://www.nature.com/articles/s41380-024-02673-8.pdf
Reference27 articles.
1. Yde Ohki CM, McNeill RV, Vernon AC, Smedler E, Michel TM, Peitz M, et al. Correspondence to “Bipolar disorder-iPSC derived neural progenitor cells exhibit dysregulation of store-operated Ca2+ entry and accelerated differentiation” by Hewitt et al. (PMID: 37402854). Mol Psychiatr. 2024. https://doi.org/10.1038/s41380-024-02602-9.
2. Hewitt, Alural T, Tilak B, Wang M, Becke J, Chartley E, et al. Bipolar disorder-iPSC derived neural progenitor cells exhibit dysregulation of store-operated Ca2+ entry and accelerated differentiation. Mol Psychiatr. 2023;28:5237–50.
3. Liou J, Kim ML, Heo WD, Jones JT, Myers JW, Ferrell JE Jr, et al. STIM is a Ca2+ sensor essential for Ca2+-store-depletion-triggered Ca2+ influx. Curr Biol. 2005;15:1235–41.
4. Brandman O, Liou J, Park WS, Meyer T. STIM2 is a feedback regulator that stabilizes basal cytosolic and endoplasmic reticulum Ca2+ levels. Cell. 2007;131:1327–39.
5. Zimmerman AJ, Hafez AK, Amoah SK, Rodriguez BA, Dell’Orco M, Lozano E, et al. Molecular Psychiatr. 2020;25:2712–27.
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