Mechanisms of ryanodine receptor 2 dysfunction in heart failure
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cardiology and Cardiovascular Medicine
Link
https://www.nature.com/articles/s41569-020-00443-x.pdf
Reference10 articles.
1. Dridi, H. et al. Intracellular calcium leak in heart failure and atrial fibrillation: a unifying mechanism and therapeutic target. Nat. Rev. Cardiol. https://doi.org/10.1038/s41569-020-0394-8 (2020).
2. Kushnir, A., Wajsberg, B. & Marks, A. R. Ryanodine receptor dysfunction in human disorders. Biochim. Biophys. Acta Mol. Cell Res. 1865, 1687–1697 (2018).
3. Wehrens, X. H. et al. Ryanodine receptor/calcium release channel PKA phosphorylation: a critical mediator of heart failure progression. Proc. Natl Acad. Sci. USA 103, 511–518 (2006).
4. Xiao, B. et al. Ser-2030, but not Ser-2808, is the major phosphorylation site in cardiac ryanodine receptors responding to protein kinase A activation upon beta-adrenergic stimulation in normal and failing hearts. Biochem. J. 396, 7–16 (2006).
5. Zhang, Y. et al. Cardiomyocyte PKA ablation enhances basal contractility while eliminates cardiac β-adrenergic response without adverse effects on the heart. Circ. Res. 124, 1760–1777 (2019).
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