Polyglutamine tracts regulate beclin 1-dependent autophagy-Reference-Cited by-同舟云学术

Polyglutamine tracts regulate beclin 1-dependent autophagy

Published:2017-04-26 Issue:7652 Volume:545 Page:108-111
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Ashkenazi Avraham,Bento Carla F.,Ricketts Thomas,Vicinanza Mariella,Siddiqi Farah,Pavel Mariana,Squitieri Ferdinando,Hardenberg Maarten C.,Imarisio Sara,Menzies Fiona M.,Rubinsztein David C.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature22078.pdf

Reference40 articles.

1. DiFiglia, M. et al. Aggregation of huntingtin in neuronal intranuclear inclusions and dystrophic neurites in brain. Science 277, 1990–1993 (1997)

2. Riess, O., Rüb, U., Pastore, A., Bauer, P. & Schöls, L. SCA3: neurological features, pathogenesis and animal models. Cerebellum 7, 125–137 (2008)

3. Imarisio, S. et al. Huntington’s disease: from pathology and genetics to potential therapies. Biochem. J. 412, 191–209 (2008)

4. Sathasivam, K. et al. Aberrant splicing of HTT generates the pathogenic exon 1 protein in Huntington disease. Proc. Natl Acad. Sci. USA 110, 2366–2370 (2013)

5. Rubinsztein, D. C. The roles of intracellular protein-degradation pathways in neurodegeneration. Nature 443, 780–786 (2006)

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