c-Abl promotes osteoblast expansion by differentially regulating canonical and non-canonical BMP pathways and p16INK4a expression-Reference-Cited by-同舟云学术

c-Abl promotes osteoblast expansion by differentially regulating canonical and non-canonical BMP pathways and p16INK4a expression

Published:2012-06-24 Issue:7 Volume:14 Page:727-737
ISSN:1465-7392
Container-title:Nature Cell Biology
language:en
Short-container-title:Nat Cell Biol

Author:

Kua Hui-Yi,Liu Huijuan,Leong Wai Fook,Li Lili,Jia Deyong,Ma Gang,Hu Yuanyu,Wang Xueying,Chau Jenny F. L.,Chen Ye-Guang,Mishina Yuji,Boast Sharon,Yeh James,Xia Li,Chen Guo-Qiang,He Lin,Goff Stephen P.,Li Baojie

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology

Link

http://www.nature.com/articles/ncb2528.pdf

Reference69 articles.

1. Harada, S. & Rodan, G. A. Control of osteoblast function and regulation of bone mass. Nature 423, 349–355 (2003).

2. Olsen, B. R., Reginato, A. M. & Wang, W. Bone development. Annu. Rev. Cell Dev. Biol. 16, 191–220 (2000).

3. Ducy, P., Schinke, T. & Karsenty, G. The osteoblast: a sophisticated fibroblast under central surveillance. Science 289, 1501–1504 (2000).

4. Canalis, E., Economides, A. N. & Gazzerro, E. Bone morphogenetic proteins, their antagonists, and the skeleton. Endocr. Rev. 24, 218–235 (2003).

5. Sharpless, N. E. & DePinho, R. A. Telomeres, stem cells, senescence, and cancer. J Clin. Invest. 113, 160–168 (2004).

Cited by 49 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. The c-Abl-RACK1-FAK signaling axis promotes renal fibrosis in mice through regulating fibroblast-myofibroblast transition;Cell Communication and Signaling;2024-04-30

2. The roles and regulatory mechanisms of TGF-β and BMP signaling in bone and cartilage development, homeostasis and disease;Cell Research;2024-01-24

3. Cuproptosis-a potential target for the treatment of osteoporosis;Frontiers in Endocrinology;2023-05-05

4. Macrophages-microenvironment crosstalk in fibrostenotic inflammatory bowel disease: from basic mechanisms to clinical applications;Expert Opinion on Therapeutic Targets;2022-11-02

5. Chi3L1 is a therapeutic target in bone metabolism and a potential clinical marker in patients with osteoporosis;Pharmacological Research;2022-10