Engineering approaches for RNA-based and cell-based osteoarthritis therapies
Author:
Publisher
Springer Science and Business Media LLC
Subject
Rheumatology
Link
https://www.nature.com/articles/s41584-023-01067-4.pdf
Reference226 articles.
1. Hsieh, J. L. et al. Intraarticular gene transfer of thrombospondin-1 suppresses the disease progression of experimental osteoarthritis. J. Orthop. Res. 28, 1300–1306 (2010).
2. Rabie, M. A. et al. Intra-articular injection of rAAV-hFGF-2 ameliorates monosodium iodoacetate-induced osteoarthritis in rats via inhibiting TLR-4 signaling and activating TIMP-1. Toxicol. Appl. Pharmacol. 459, 116361 (2023).
3. Chen, B., Qin, J., Wang, H., Magdalou, J. & Chen, L. Effects of adenovirus-mediated bFGF, IL-1Ra and IGF-1 gene transfer on human osteoarthritic chondrocytes and osteoarthritis in rabbits. Exp. Mol. Med. 42, 684–695 (2010).
4. Frisbie, D. D., Ghivizzani, S. C., Robbins, P. D., Evans, C. H. & McIlwraith, C. W. Treatment of experimental equine osteoarthritis by in vivo delivery of the equine interleukin-1 receptor antagonist gene. Gene Ther. 9, 12–20 (2002).
5. Schrenker, S. et al. In vivo rAAV-mediated human TGF-β overexpression reduces perifocal osteoarthritis and improves osteochondral repair in a large animal model at one year. Osteoarthritis Cartilage 31, 467–481 (2023).
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