Increased endothelin-1 and decreased adrenomedullin gene expression in the stenotic tissue of congenital pelvi-ureteric junction obstruction in children
Author:
Publisher
Wiley
Subject
Urology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1464-410x.2001.02173.x/fullpdf
Reference25 articles.
1. Abnormal innervation and altered nerve growth factor messenger ribonucleic acid expression in ureteropelvic junction obstruction;Wang;J Urol,1995
2. Peptidergic innervation of the normal and obstructed human pyeloureteral junctions;Tainio;Urol Int,1992
3. Renal tissue endothelin in long term complete ureteric obstruction in the young rats;Josephson;Urol Int,1994
4. Contractile effects of endothelin-1 and localization of endothelin binding sites in rabbit lower urinary tract smooth muscle;Garcia-Pascual;Acta Physiol Scand,1990
5. Endothelin stimulates c-fos and c-myc expression and proliferation of vascular smooth muscle cells;Komuro;FEBS Lett,1988
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1. A systematic review of underlying genetic factors associated with ureteropelvic junction obstruction in stenotic human tissue;Journal of Pediatric Urology;2022-10
2. Uric acid causes kidney injury through inducing fibroblast expansion, Endothelin-1 expression, and inflammation;BMC Nephrology;2017-10-31
3. The molecular biology of pelvi-ureteric junction obstruction;Pediatric Nephrology;2017-03-13
4. Role of Urinary Levels of Endothelin-1, Monocyte Chemotactic Peptide-1, andN-Acetyl Glucosaminidase in Predicting the Severity of Obstruction in Hydronephrotic Neonates;Korean Journal of Urology;2014
5. Thickness of the renal pelvis smooth muscle indicates the postoperative course of ureteropelvic junction obstruction treatment;Actas Urológicas Españolas (English Edition);2011-11
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