INK4A/ARF deletions are acquired at relapse in childhood acute lymphoblastic leukaemia: a paired study on 25 patients using real-time polymerase chain reaction
Author:
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1365-2141.2001.02729.x/fullpdf
Reference36 articles.
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2. Cytogenetic studies on acute lymphoblastic leukemia in relapse;Berger;Cancer and Genetics,1988
3. Minimal residual disease in childhood acute lymphoblastic leukemia: analysis of patients in continuous complete remission or with consecutive relapse;Biondi;Leukemia,1992
4. Exon 5 mutations in the p53 gene in relapsed childhood acute lymphoblastic leukemia;Blau;Leukemia Reseach,1997
5. Frequency of homozygous deletion at p16/CDKN2 in primary human tumours;Cairns;Nature Genetics,1995
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1. The Yin and Yang-Like Clinical Implications of the CDKN2A/ARF/CDKN2B Gene Cluster in Acute Lymphoblastic Leukemia;Genes;2021-01-09
2. Constitutive Ras signaling and Ink4a/Arf inactivation cooperate during the development of B-ALL in mice;Blood Advances;2017-11-21
3. Routine application of a novel MLPA-based first-line screening test uncovers clinically relevant copy number aberrations in haematological malignancies undetectable by conventional cytogenetics;Hematology;2013-11-25
4. Prognostic Significance of p14ARF, p15INK4b, and p16INK4a Inactivation in Malignant Peripheral Nerve Sheath Tumors;Clinical Cancer Research;2011-01-24
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