PERSISTENCE OF THE NITRIC OXIDE-DEPENDENT VASODILATORPATHWAY OF CEREBRAL VESSELS AFTEREXPERIMENTAL SUBARACHNOID HEMORRHAGE
Author:
Affiliation:
1. Department of Neurosurgery, Johann Wolfgang Goethe-University, Frankfurt, Germany
2. Institute for Neuroradiology, Johann Wolfgang Goethe-University, Frankfurt, Germany
3. Department of Neurosurgery, Hospital Duisburg-North, Duisburg, Germany
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Neurology (clinical),Surgery
Link
http://academic.oup.com/neurosurgery/article-pdf/60/1/179/24534822/00006123-200701000-00020.pdf
Reference62 articles.
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2. Effects of a nitric oxide donor on and correlation of changes in cyclic nucleotide levels with experimental vasospasm;Aihara;Neurosurgery,2003
3. Functional changes in human pial arteries (300 to 900 micrometer ID) within 48 hours of aneurysmal subarachnoid hemorrhage;Bevan;Stroke,1998
4. Increased nitrovasodilator sensitivity in endothelial nitric oxide synthase knockout mice: Role of soluble guanylyl cyclase;Brandes;Hypertension,2000
5. Controlled release of a nitric oxide donor for the prevention of delayed cerebral vasospasm following experimental subarachnoid hemorrhage in nonhuman primates;Clatterbuck;J Neurosurg,2005
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