Differential Effects of Tumor Necrosis Factor-Alpha on Proliferation, Cell Surface Antigen Expression, and Cytokine Interactions in Malignant Gliomas

Author:

Chen Thomas C.1,Hinton David R.12,Apuzzo Michael L. J.1,Hofman Florence M.2

Affiliation:

1. Departments of Neurological Surgery, Los Angeles County Hospital/University of Southern California Medical Center and University of Southern California School of Medicine, Los Angeles, California

2. Pathology, Los Angeles County Hospital/University of Southern California Medical Center and University of Southern California School of Medicine, Los Angeles, California

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Clinical Neurology,Surgery

Reference54 articles.

1. IL-6 inhibits lipopolysaccharide-induced tumor necrosis factor production in cultured human monocytes, U937 cells, and in mice;Aderka;J Immunol,1989

2. Biology of multifunctional cytokines: IL-6 and related molcules (IL-1 and TNF);Akira;FASEB J,1990

3. Immunological aspects of intrinsic glial tumors;Apuzzo;J Neurosurg,1981

4. Tumor necrosis factor-α enhances interferon-γ-mediated class II antigen expression on astrocytes;Benveniste;J Neuroimmunol,1989

5. Tumor necrosis factor production and receptor expression by a human malignant glioma cell line, D54-MG;Bethea;J Neuroimmunol,1990

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