Hydrolytic Activity of Mitochondrial F1FO-ATP Synthase as a Target for Myocardial Ischemia–Reperfusion Injury: Discovery and In Vitro and In Vivo Evaluation of Novel Inhibitors-Reference-Cited by-同舟云学术

Hydrolytic Activity of Mitochondrial F₁F_O-ATP Synthase as a Target for Myocardial Ischemia–Reperfusion Injury: Discovery and In Vitro and In Vivo Evaluation of Novel Inhibitors

Published:2023-11-09 Issue:22 Volume:66 Page:15115-15140
ISSN:0022-2623
Container-title:Journal of Medicinal Chemistry
language:en
Short-container-title:J. Med. Chem.

Author:

Nikolaou Panagiota-Efstathia¹,Lambrinidis George²^ORCID,Georgiou Maria²,Karagiannis Dimitrios²^ORCID,Efentakis Panagiotis¹,Bessis-Lazarou Pavlos¹,Founta Konstantina¹,Kampoukos Stavros²,Konstantin Vasilis²,Palmeira Carlos M.³^ORCID,Davidson Sean M.⁴,Lougiakis Nikolaos²,Marakos Panagiotis²,Pouli Nicole²,Mikros Emmanuel²⁵^ORCID,Andreadou Ioanna¹

Affiliation:

1. Laboratory of Pharmacology, Faculty of Pharmacy, National and Kapodistrian University of Athens, 15771 Athens, Greece

2. Department of Pharmaceutical Chemistry, Faculty of Pharmacy, National and Kapodistrian University of Athens, 15771 Athens, Greece

3. Department of Life Sciences, University of Coimbra and Center for Neurosciences and Cell Biology, University of Coimbra, 3004-504 Coimbra, Portugal

4. The Hatter Cardiovascular Institute, University College London, 67 Chenies Mews, WC1E 6HX London, United Kingdom

5. Athena Research and Innovation Center in Information Communication & Knowledge Technologies, 15125 Marousi, Greece

Funder

British Heart Foundation

European Cooperation in Science and Technology

Publisher

American Chemical Society (ACS)

Subject

Drug Discovery,Molecular Medicine

Link

https://pubs.acs.org/doi/pdf/10.1021/acs.jmedchem.3c01048

Reference84 articles.

1. Cardioprotection by H2S Donors: Nitric Oxide-Dependent and -Independent Mechanisms

2. Cardiac metabolism as a driver and therapeutic target of myocardial infarction

4. Myocardial ischaemia–reperfusion injury and cardioprotection in perspective

5. The quantification of infarct size

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