Aβ Amyloid Fibers Drastically Alter the Topography and Mechanical Properties of Lipid Membranes-Reference-Cited by-同舟云学术

Aβ Amyloid Fibers Drastically Alter the Topography and Mechanical Properties of Lipid Membranes

Published:2023-12-11 Issue:51 Volume:39 Page:18923-18934
ISSN:0743-7463
Container-title:Langmuir
language:en
Short-container-title:Langmuir

Author:

Alvarez Alain Bolaño¹²³,Rodríguez Pablo E. A.⁴,Fidelio Gerardo D.¹²^ORCID,Caruso Benjamín⁵⁶^ORCID

Affiliation:

1. Departamento de Química Biológica Ranwel Caputto, Facultad de Ciencias Químicas, Universidad Nacional de Córdoba, Córdoba CP5000, Argentina

2. Centro de Investigaciones en Química Biológica de Córdoba, CIQUIBIC, CONICET-Universidad Nacional de Córdoba, Córdoba CP5000, Argentina

3. Department of Dermatology and Venerology, Aalborg University Hospital, Aalborg, DK-9000 Denmark

4. Ministerio de Ciencia y Tecnología de Córdoba, Córdoba CP5000, Argentina

5. Cátedra de Química Biológica, Departamento de Química, Facultad de Ciencias Exactas, Físicas y Naturales, Universidad Nacional de Córdoba, Córdoba CP5000, Argentina

6. Instituto de Investigaciones Biológicas y Tecnológicas (IIBYT), CONICET, Universidad Nacional de Córdoba, Córdoba CP5000, Argentina

Funder

Ministerio de Ciencia y Tecnolog?a, Gobierno de la Provincia de C?rdoba

Consejo Nacional de Investigaciones Cient?ficas y T?cnicas

Secretaria de Ciencia y Tecnolog?a - Universidad Nacional de C?rdoba

Fondo para la Investigaci?n Cient?fica y Tecnol?gica

Publisher

American Chemical Society (ACS)

Subject

Electrochemistry,Spectroscopy,Surfaces and Interfaces,Condensed Matter Physics,General Materials Science

Link

https://pubs.acs.org/doi/pdf/10.1021/acs.langmuir.3c02831

Reference40 articles.

1. Membrane-mediated fibrillation and toxicity of the tau hexapeptide PHF6

2. Fibrillar and Nonfibrillar Amyloid Beta Structures Drive Two Modes of Membrane-Mediated Toxicity

3. Alzheimer’s Disease as a Membrane Disorder: Spatial Cross-Talk Among Beta-Amyloid Peptides, Nicotinic Acetylcholine Receptors and Lipid Rafts

4. Publisher Correction: A dopamine metabolite stabilizes neurotoxic amyloid-β oligomers

5. Neurotoxic Soluble Amyloid Oligomers Drive Alzheimer’s Pathogenesis and Represent a Clinically Validated Target for Slowing Disease Progression