Cellular and molecular mechanisms of Notch signal in pulmonary microvascular endothelial cells after acute lung injury
Author:
Affiliation:
1. Hangzhou Red Cross Hospital, China
Publisher
FapUNIFESP (SciELO)
Subject
Cell Biology,General Pharmacology, Toxicology and Pharmaceutics,General Medicine,Immunology,Physiology,Biochemistry,General Neuroscience,Biophysics,General Medicine
Link
http://www.scielo.br/scielo.php?script=sci_pdf&pid=S0100-879X2023000100698&tlng=en
Reference29 articles.
1. Depletion of circulating monocytes suppresses IL-17 and HMGB1 expression in mice with LPS-induced acute lung injury;Jiang Z;Am J Physiol Lung Cell Mol Physiol,2017
2. Th17/Treg imbalance: Implications in Llng inflammatory diseases;Thomas R;Int J Mol Sci,2023
3. DAPT reverses the Th17/Treg imbalance in experimental autoimmune uveitis in vitro via inhibiting Notch signaling pathway;Yin X;Int Immunopharmacol,2020
4. Inactivation of Notch signaling reverses the Th17/Treg imbalance in cells from patients with immune thrombocytopenia;Yu S;Lab Invest,2015
5. The ratio of Th17/Treg cells as a risk indicator in early acute respiratory distress syndrome;Yu ZX;Crit Care,2015
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