Acute Myeloid Leukemia Chemo-Resistance Is Mediated by E-selectin Receptor CD162 in Bone Marrow Niches-Reference-Cited by-同舟云学术

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Acute Myeloid Leukemia Chemo-Resistance Is Mediated by E-selectin Receptor CD162 in Bone Marrow Niches

Published:2020-07-24 Issue: Volume:8 Page:
ISSN:2296-634X
Container-title:Frontiers in Cell and Developmental Biology
language:
Short-container-title:Front. Cell Dev. Biol.

Author:

Erbani Johanna,Tay Joshua,Barbier Valerie,Levesque Jean-Pierre,Winkler Ingrid G.

Funder

National Health and Medical Research Council

University of Queensland

Mater Foundation

Publisher

Frontiers Media SA

Subject

Cell Biology,Developmental Biology

Reference73 articles.

1. The 130-kDa glycoform of CD43 functions as an E-selectin ligand for activated Th1 cells in vitro and in delayed-type hypersensitivity reactions in vivo.;Alcaide;J. Invest. Dermatol.,2007

2. The P-selectin glycoprotein ligand functions as a common human leukocyte ligand for P- and E-selectins.;Asa;J. Biol. Chem.,1995

3. P-selectin glycoprotein ligand regulates the interaction of multiple myeloma cells with the bone marrow microenvironment.;Azab;Blood,2012

4. Endothelial E-selectin inhibition improves acute myeloid leukaemia therapy by disrupting vascular niche-mediated chemoresistance.;Barbier;Nat. Commun.,2020

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