Myocyte [Na+]i Dysregulation in Heart Failure and Diabetic Cardiomyopathy
Author:
Publisher
Frontiers Media SA
Subject
Physiology (medical),Physiology
Reference86 articles.
1. Inhibition of the Na+/H+ exchanger attenuates the deterioration of ventricular function during pacing-induced heart failure in rabbits.;Aker;Cardiovasc. Res.,2004
2. Intracellular sodium increase and susceptibility to ischaemia in hearts from type 2 diabetic db/db mice.;Anzawa;Diabetologia,2006
3. Increased Na+/H+-exchange activity is the cause of increased [Na+]i and underlies disturbed calcium handling in the rabbit pressure and volume overload heart failure model.;Baartscheer;Cardiovasc. Res.,2003
4. Chronic inhibition of Na+/H+-exchanger attenuates cardiac hypertrophy and prevents cellular remodeling in heart failure.;Baartscheer;Cardiovasc. Res.,2005
5. Empagliflozin decreases myocardial cytoplasmic Na+ through inhibition of the cardiac Na+/H+ exchanger in rats and rabbits.;Baartscheer;Diabetologia,2017
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