Macrophage Regnase-1 Deletion Deteriorates Liver Ischemia/Reperfusion Injury Through Regulation of Macrophage Polarization
Author:
Publisher
Frontiers Media SA
Subject
Physiology (medical),Physiology
Reference33 articles.
1. A translational silencing function of MCPIP1/Regnase-1 specified by the target site context.;Behrens;Nucleic Acids Res.,2018
2. MCPIP1 contributes to the inflammatory response of UVB-treated keratinocytes.;Bugara;J. Dermatol. Sci.,2017
3. VEGF-C attenuates ischemia reperfusion injury of liver graft in rats.;Cheng;Transpl. Immunol.,2019
4. Regnase-1 and roquin nonredundantly regulate Th1 differentiation causing cardiac inflammation and fibrosis.;Cui;J. Immunol.,2017
5. Induction of an inflammatory response in primary hepatocyte cultures from mice.;Czaya;Jove J. Vis. Exper.,2017
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1. In-vivo screening implicates endoribonuclease Regnase-1 in modulating senescence-associated lysosomal changes;GeroScience;2023-08-29
2. New Insights into Baicalein’s Effect on Chlorpyrifos-Induced Liver Injury in Carp: Involving Macrophage Polarization and Pyropto sis;Journal of Agricultural and Food Chemistry;2023-02-27
3. Gut-Derived Exosomes Induce Liver Injury After Intestinal Ischemia/Reperfusion by Promoting Hepatic Macrophage Polarization;Inflammation;2022-06-14
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