New Insights Into the Role and Mechanism of Partial Epithelial-Mesenchymal Transition in Kidney Fibrosis
Author:
Funder
National Natural Science Foundation of China
National Institutes of Health
Publisher
Frontiers Media SA
Subject
Physiology (medical),Physiology
Reference110 articles.
1. Proximal tubular cells promote fibrogenesis by TGF-beta1-mediated induction of peritubular myofibroblasts.;Abbate;Kidney Int.,2002
2. Sonic hedgehog-mediated epithelial-mesenchymal transition in renal tubulointerstitial fibrosis.;Bai;Int. J. Mol. Med.,2016
3. Resveratrol inhibits epithelial-mesenchymal transition and renal fibrosis by antagonizing the hedgehog signaling pathway.;Bai;Bioche. Pharmacol.,2014
4. Methylation determines fibroblast activation and fibrogenesis in the kidney.;Bechtel;Nat. Med.,2010
5. Epithelial Notch signaling regulates interstitial fibrosis development in the kidneys of mice and humans.;Bielesz;J. Clin. Invest.,2010
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