Renal Cell Carcinoma in von Hippel–Lindau Disease—From Tumor Genetics to Novel Therapeutic Strategies
Author:
Publisher
Frontiers Media SA
Subject
Pediatrics, Perinatology and Child Health
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2. Zur Frage der Angiomatosis Retinae und Ihrer Hirncomplikation;Lindau;Acta Ophthalmol,1927
3. Intra-ocular growths;Collins;Trans Ophthalmol Soc U K,1894
4. Clustering of features of von Hippel-Lindau syndrome: evidence for a complex genetic locus;Neumann;Lancet,1991
5. Germline mutations in the von Hippel-Lindau disease tumor suppressor gene: correlations with phenotype;Chen;Hum Mutat,1995
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2. FOXA2 activates HIF2α expression to promote tumor progression and is regulated by the E3 ubiquitin ligase VHL in renal cell carcinoma;Journal of Biological Chemistry;2024-01
3. Sunitinib Treatment of VHL C162F Cells Slows Down Proliferation and Healing Ability via Downregulation of ZHX2 and Confers a Mesenchymal Phenotype;Cancers;2023-12-20
4. Differential expression of HIF1A and its downstream target VEGFA in the main subtypes of renal cell carcinoma and their impact on patient survival;Frontiers in Oncology;2023-11-20
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