A Novel 3D Osteoblast and Osteocyte Model Revealing Changes in Mineralization and Pro-osteoclastogenic Paracrine Signaling During Estrogen Deficiency-Reference-Cited by-同舟云学术

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A Novel 3D Osteoblast and Osteocyte Model Revealing Changes in Mineralization and Pro-osteoclastogenic Paracrine Signaling During Estrogen Deficiency

Published:2020-06-10 Issue: Volume:8 Page:
ISSN:2296-4185
Container-title:Frontiers in Bioengineering and Biotechnology
language:
Short-container-title:Front. Bioeng. Biotechnol.

Author:

Naqvi Syeda Masooma,Panadero Pérez Juan Alberto,Kumar Vatsal,Verbruggen Anneke S. K.,McNamara Laoise M.

Funder

Irish Research Council

Science Foundation Ireland

Publisher

Frontiers Media SA

Subject

Biomedical Engineering,Histology,Bioengineering,Biotechnology

Reference157 articles.

1. Oscillating fluid flow regulates gap junction communication in osteocytic MLO-Y4 cells by an ERK1/2 MAP kinase-dependent mechanism.;Alford;Bone,2003

2. Inhibition of osteoclastogenesis by mechanically stimulated osteoblasts is attenuated during estrogen deficiency.;Allison;Am. J. Physiol. Cell. Physiol.,2019

3. Mechanically induced osteogenic differentiation–the role of RhoA, ROCKII and cytoskeletal dynamics.;Arnsdorf;J. Cell. Sci.,2009

4. Vitamin K promotes mineralization, osteoblast-to-osteocyte transition, and an anticatabolic phenotype by {gamma}-carboxylation-dependent and -independent mechanisms.;Atkins;Am. J. Physiol. Cell. Physiol.,2009

5. Different responsiveness to mechanical stress of bone cells from osteoporotic versus osteoarthritic donors.;Bakker;Osteoporos. Int.,2006

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5. Postmenopausal osteoporosis coexisting with sarcopenia: the role and mechanisms of estrogen;Journal of Endocrinology;2023-07-31

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