Endoplasmic Reticulum Stress Activated by Androgen Enhances Apoptosis of Granulosa Cells via Induction of Death Receptor 5 in PCOS-Reference-Cited by-同舟云学术

Endoplasmic Reticulum Stress Activated by Androgen Enhances Apoptosis of Granulosa Cells via Induction of Death Receptor 5 in PCOS

Published:2018-11-12 Issue:1 Volume:160 Page:119-132
ISSN:1945-7170
Container-title:Endocrinology
language:en
Short-container-title:

Author:

Azhary Jerilee M K¹,Harada Miyuki¹^ORCID,Takahashi Nozomi¹,Nose Emi¹,Kunitomi Chisato¹,Koike Hiroshi¹,Hirata Tetsuya¹,Hirota Yasushi¹,Koga Kaori¹,Wada-Hiraike Osamu¹,Fujii Tomoyuki¹,Osuga Yutaka¹

Affiliation:

1. Department of Obstetrics and Gynecology, Faculty of Medicine, The University of Tokyo, Tokyo, Japan

Funder

Japan Agency for Medical Research and Development

Takeda Science Foundation

Publisher

The Endocrine Society

Subject

Endocrinology

Link

http://academic.oup.com/endo/article-pdf/160/1/119/27239799/en.2018-00675.pdf

Reference72 articles.

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2. Scientific statement on the diagnostic criteria, epidemiology, pathophysiology, and molecular genetics of polycystic ovary syndrome;Dumesic;Endocr Rev,2015

3. Follicular hyperandrogenism and insulin resistance in polycystic ovary syndrome patients with normal circulating testosterone levels;Li;J Biomed Res,2019

4. Polycystic ovary syndrome;McCartney;N Engl J Med,2016

5. The pathogenesis of polycystic ovary syndrome (PCOS): the Hypothesis of PCOS as functional ovarian hyperandrogenism revisited;Rosenfield;Endocr Rev,2016

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