L-Type Calcium Channels in Insulin-Secreting Cells: Biochemical Characterization and Phosphorylation in RINm5F Cells

Author:

Safayhi Hasan1,Haase Hannelore2,Kramer Ursel1,Bihlmayer Andrea1,Roenfeldt Monika1,Ammon Hermann P.T.1,Froschmayr Monika3,Cassidy Tara N.3,Morano Ingo2,Ahlijanian Michael K.4,Striessnig Jörg3

Affiliation:

1. Pharmazeutisches Institut, Lehrstuhl Pharmakologie (H.S., U.K., A.B., M.R., H.P.T.A.) Universität Tübingen D-72076 Tübingen, Germany

2. Max Delbrück Centrum für Molekulare Medizin (H.H., I.M.) Cardiology D-13125 Berlin, Germany

3. Institut für Biochemische Pharmakologie (J.S., M.F., T.N.C.) Universität Innsbruck A-6020 Innsbruck, Austria

4. Pfizer (M.K.A.) Central Research Division Groton, Connecticut 06340

Publisher

The Endocrine Society

Subject

Endocrinology,Molecular Biology,General Medicine

Reference65 articles.

1. Stimulus-secretion coupling in pancreaticβ cells;Ashcroft;J Cell Biochem,1994

2. Modulation of dihydropyridine-sensitive Ca2+ channels by glucose metabolism in mouse pancreatic beta-cells.;Smith;Nature,1989

3. Catecholamine modulation of calcium currents in clonal pancreatic beta-cells;Keahey;Am J Physiol,1989

4. Somatostatin inhibits insulin secretion by a G-protein-mediated decrease in Ca2+ entry through voltage-dependent Ca2+ channels in the beta cell.;Hsu;J Biol Chem,1991

5. Inhibition of L-type calcium channels by internal GTP[ gamma-S] in mouse pancreatic β cells.;Ämmälä;Pflügers Arch,1992

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