Targeting Elevated GnRH Pulsatility to Treat Polycystic Ovary Syndrome

Author:

Abbara Ali1ORCID,Dhillo Waljit S1ORCID

Affiliation:

1. Section of Endocrinology and Investigative Medicine, Imperial College London, Hammersmith Hospital, London, W12 0NN, UK

Funder

National Institute for Health Research

Imperial Biomedical Research Centre

Medical Research Council

Biotechnology and Biological Sciences Research Council

Publisher

The Endocrine Society

Subject

Biochemistry, medical,Clinical Biochemistry,Endocrinology,Biochemistry,Endocrinology, Diabetes and Metabolism

Reference8 articles.

1. Clinical and biochemical discriminants between functional hypothalamic amenorrhoea (FHA) and polycystic ovary syndrome (PCOS);Phylactou;Clin Endocrinol (Oxf).,2020

2. Neuroendocrine androgen action is a key extraovarian mediator in the development of polycystic ovary syndrome;Caldwell;Proc Natl Acad Sci U S A.,2017

3. Polycystic οvary syndrome revisited: an interactions network approach;Geronikolou;Eur J Clin Invest.,2021

4. Neurokinin B receptor antagonism in women with polycystic ovary syndrome: a randomized, placebo-controlled trial;George;J Clin Endocrinol Metab.,2016

5. The NK3 receptor antagonist ESN364 suppresses sex hormones in men and women;Fraser;J Clin Endocrinol Metab.,2016

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