Mutations in fetal genes involved in innate immunity and host defense against microbes increase risk of preterm premature rupture of membranes (PPROM)

Author:

Modi Bhavi P.1,Teves Maria E.2,Pearson Laurel N.3,Parikh Hardik I.4,Haymond-Thornburg Hannah2,Tucker John L.2,Chaemsaithong Piya5,Gomez-Lopez Nardhy5678,York Timothy P.12,Romero Roberto5678,Strauss Jerome F.12ORCID

Affiliation:

1. Department of Human and Molecular Genetics; Virginia Commonwealth University; Richmond Virginia

2. Department of Obstetrics and Gynecology; Virginia Commonwealth University; Richmond Virginia

3. Department of Anthropology; Pennsylvania State University; University Park Pennsylvania

4. Department of Microbiology and Immunology; Virginia Commonwealth University; Richmond Virginia

5. Perinatology Research Branch; Eunice Kennedy Shriver National Institute for Child Health and Human Development; NIH; Detroit Michigan

6. Department of Obstetrics and Gynecology; University of Michigan; Ann Arbor Michigan

7. Department of Epidemiology and Biostatistics; Michigan State University; East Lansing Michigan

8. Center for Molecular Medicine and Genetics; Wayne State University; Detroit Michigan

Funder

National Institute on Minority Health and Health Disparities

Eunice Kennedy Shriver National Institute of Child Health and Human Development

National Institutes of Health

National Institutes of Health, Department of Health and Human Services (NICHD/NIH)

Publisher

Wiley

Subject

Genetics (clinical),Genetics,Molecular Biology

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