Myelin as an inflammatory mediator: Myelin interactions with complement, macrophages, and microglia in spinal cord injury

Author:

Kopper Timothy J.1ORCID,Gensel John C.1

Affiliation:

1. Spinal Cord and Brain Injury Research Center; Department of Physiology, College of Medicine, University of Kentucky; Lexington Kentucky

Funder

NIH

National Institute of Neurological Disorders and Stroke

Spinal Cord and Brain Injury Research Center at the University of Kentucky

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience

Reference81 articles.

1. Activation of complement pathways after contusion-induced spinal cord injury;Anderson;Journal of Neurotrauma,2004

2. Complement biosynthesis in the central nervous system;Barnum;Critical Reviews in Oral Biology and Medicine,1995

3. MR-pathologic comparisons of wallerian degeneration in spinal cord injury;Becerra;American Journal of Neuroradiology,1995

4. Quantitative analysis of cellular inflammation after traumatic spinal cord injury: Evidence for a multiphasic inflammatory response in the acute to chronic environment;Beck;Brain,2010

5. Myelin alters the inflammatory phenotype of macrophages by activating PPARs;Bogie;Acta Neuropathologica Communications,2013

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