Elevated expression of long intergenic non-coding RNA HOTAIR in a basal-like variant of MCF-7 breast cancer cells

Author:

Zhuang Yan1,Nguyen Hong T.1,Burow Matthew E.1,Zhuo Ying2,El-Dahr Samir S.3,Yao Xiao2,Cao Subing4,Flemington Erik K.4,Nephew Kenneth P.5,Fang Fang5,Collins-Burow Bridgette1,Rhodes Lyndsay V.1,Yu Qiang6,Jayawickramarajah Janarthanan7,Shan Bin8

Affiliation:

1. Department of Medicine; Tulane University School of Medicine; New Orleans Louisiana

2. Kadlec Regional Medical Center; Richland Washington

3. Department of Pediatrics; Tulane University School of Medicine; New Orleans Louisiana

4. Department of Pathology; Tulane University School of Medicine; New Orleans Louisiana

5. Department of Medical Sciences; Indiana University School of Medicine; Bloomington Indiana

6. Genome Institute of Singapore; Singapore Singapore

7. Department of Chemistry; Tulane University; New Orleans Louisiana

8. Washington State University at Spokane; Spokane Washington

Funder

Washington State University Startup Fund

National Institutes of Health

Publisher

Wiley

Subject

Cancer Research,Molecular Biology

Reference65 articles.

1. Sensitization of apoptotically-resistant breast carcinoma cells to TNF and TRAIL by inhibition of p38 mitogen-activated protein kinase signaling;Weldon;Int J Oncol,2004

2. Identification of mitogen-activated protein kinase kinase as a chemoresistant pathway in MCF-7 cells by using gene expression microarray;Weldon;Surgery,2002

3. NF-kappa B-mediated chemoresistance in breast cancer cells;Weldon;Surgery,2001

4. Altered death receptor signaling promotes epithelial-to-mesenchymal transition and acquired chemoresistance;Antoon;Sci Rep,2012

5. Targeting NFkB mediated breast cancer chemoresistance through selective inhibition of sphingosine kinase-2;Antoon;Cancer Biol Ther,2011

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