VEGF‐A promotes angiogenesis after acute myocardial infarction through increasing ROS production and enhancing ER stress‐mediated autophagy

Author:

Zou Jiang12,Fei Qin12,Xiao Hui12,Wang Hao12,Liu Ke12,Liu Meidong12,Zhang Huali12,Xiao Xianzhong12,Wang Kangkai123ORCID,Wang Nian12ORCID

Affiliation:

1. Department of Pathophysiology School of Basic Medical Science, Central South University Changsha Hunan China

2. Key Laboratory of Sepsis Translational Medicine of Hunan, Central South University Changsha Hunan China

3. Department of Laboratory Animals Hunan Key Laboratory of Animal Models for Human Diseases, Xiangya School of Medicine, Central South University Changsha Hunan China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Reference57 articles.

1. Effect of autophagy on myocardial infarction and its mechanism;Aisa Z.;European Review for Medical and Pharmacological Sciences,2017

2. Effect of tauroursodeoxycholic acid on PUFA levels and inflammation in an animal and cell model of hepatic endoplasmic reticulum stress

3. Prevalence and prevention of cardiovascular disease and diabetes mellitus

4. ER Stress and Angiogenesis

5. Coronary artery ectasia as a culprit for acute myocardial infarction: Review of pathophysiology and management;Boles U.;Anadolu kardiyoloji dergisi: AKD = the Anatolian journal of cardiology,2013

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