Effects of CXCL4/CXCR3 on the lipopolysaccharide‐induced injury in human umbilical vein endothelial cells
Author:
Affiliation:
1. Faculty of Anesthesiology Changhai Hospital, Naval Medical University Shanghai China
2. Department of Intensive Care Unit Shanghai General Hospital of Chinese Armed Police Force China
Funder
National Natural Science Foundation of China
Publisher
Wiley
Subject
Cell Biology,Clinical Biochemistry,Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.28803
Reference33 articles.
1. LPS-induced MCP-1 expression in human microvascular endothelial cells is mediated by the tyrosine kinase, Pyk2 via the p38 MAPK/NF-κB-dependent pathway
2. Naringin inhibits lipopolysaccharide-induced damage in human umbilical vein endothelial cells via attenuation of inflammation, apoptosis and MAPK pathways
3. Claudin-4 is required for modulation of paracellular permeability by muscarinic acetylcholine receptor in epithelial cells
4. Inhibition of Human Umbilical Vein Endothelial Cell Proliferation by the CXC Chemokine, Platelet Factor 4 (PF4), Is Associated With Impaired Downregulation of p21Cip1/WAF1
5. Cigarette Smoke–Induced CXCR3 Receptor Up-Regulation Mediates Endothelial Apoptosis
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