Pyk2 and Src Mediate Signaling to CCL18-Induced Breast Cancer Metastasis

Author:

Li Hai-Yan12,Cui Xiu-Ying1,Wu Wei1,Yu Feng-Yan1,Yao He-Rui1,Liu Qiang1,Song Er-Wei1,Chen Jing-Qi13

Affiliation:

1. Breast Cancer Center; Sun-Yat-Sen Memorial Hospital; Sun-Yat-Sen University; Guangzhou 510120 People's Republic of China

2. Department of Breast and Thyroid Surgery; The 6th Affriciated Hospital of Sun Yat-Sen University; Sun-Yat-Sen University; Guangzhou 510120 People's Republic of China

3. Department of Medical Oncology; No. 2 Affiliated Hospital; Guangzhou Medical College; Guangzhou 510260 People's Republic of China

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference32 articles.

1. Identification of a new Pyk2 target protein with Arf-GAP activity;Andreev;Mol Cell Biol,1999

2. Postsynaptic clustering and activation of Pyk2 by PSD-95;Bartos;J Neurosci,2010

3. Integrin beta5 contributes to the tumorigenic potential of breast cancer cells through the Src-FAK and MEK-ERK signaling pathways;Bianchi-Smiraglia;Oncogene,2012

4. High expression of proline-rich tyrosine kinase2 is associated with poor survival of hepatocellular carcinoma via regulating phosphatidylinositol 3-kinase/AKT pathway;Cao;Ann Surg Oncol,2012

5. CCL18 from tumor-associated macrophages promotes breast cancer metastasis via PITPNM3;Chen;Cancer Cell,2011

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