Insulin acutely activates metabolism of primary human monocytes and promotes a proinflammatory phenotype-Reference-Cited by-同舟云学术

Insulin acutely activates metabolism of primary human monocytes and promotes a proinflammatory phenotype

Published:2021-01-21 Issue:5 Volume:110 Page:885-891
ISSN:0741-5400
Container-title:Journal of Leukocyte Biology
language:en
Short-container-title:J Leukoc Biol

Author:

Ratter Jacqueline M.¹²³⁴,Heck Julia I. P.¹,Rooijackers Hanne M. M.¹,Jansen Henry J.¹,Poppel Pleun C. M.¹,Tack Cees J.¹,Stienstra Rinke¹²^ORCID

Affiliation:

1. Department of Internal Medicine Radboud University Medical Center Nijmegen The Netherlands

2. Nutrition, Metabolism and Genomics Group, Division of Human Nutrition and Health Wageningen University and Research Wageningen The Netherlands

3. Institute for Clinical Diabetology, German Diabetes Center Leibniz Center for Diabetes Research at Heinrich Heine University Düsseldorf Düsseldorf Germany

4. German Center for Diabetes Research (DZD) Düsseldorf Germany

Funder

National Science Foundation

Publisher

Wiley

Subject

Cell Biology,Immunology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/JLB.3AB0120-019RR

Reference27 articles.

1. Insulin binding, insulin degradation and glucose metabolism in human monocytes

2. Insulin Receptor-Mediated Stimulation Boosts T Cell Immunity during Inflammation and Infection

3. Myeloid Cell-Restricted Insulin Receptor Deficiency Protects Against Obesity-Induced Inflammation and Systemic Insulin Resistance

4. Downregulation of macrophage Irs2 by hyperinsulinemia impairs IL-4-indeuced M2a-subtype macrophage activation in obesity

5. Palmitate and insulin synergistically induce IL-6 expression in human monocytes

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