Lack of ifn-?-mediated induction of the class II transactivator (CIITA) through promoter methylation is predominantly found in developmental tumor cell lines-Reference-Cited by-同舟云学术

Lack of ifn-?-mediated induction of the class II transactivator (CIITA) through promoter methylation is predominantly found in developmental tumor cell lines

Published:2002-01-15 Issue:4 Volume:97 Page:501-507
ISSN:0020-7136
Container-title:International Journal of Cancer
language:en
Short-container-title:Int. J. Cancer

Author:

van der Stoep Nienke,Biesta Paula,Quinten Edwin,van den Elsen Peter J.

Publisher

Wiley

Subject

Cancer Research,Oncology

Reference55 articles.

1. Complementation cloning of an MHC class II transactivator mutated in hereditary MHC class II deficiency (or bare lymphocyte syndrome)

2. Molecular analysis of G1B and G3A IFNγ mutants reveals that defects in CIITA or RFX result in defective class II MHC and li gene induction

3. Class II transactivator regulates the expression of multiple genes involved in antigen presentation.

4. The two novel MHC class II transactivators RFX5 and CIITA both control expression of HLA-DM genes

5. Activation of class II MHC genes requires both the X ☐ region and the class II transactivator (CIITA)

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