Acute myeloid leukemia with inv(16)(p13.1q22), abnormal eosinophils, and absence of peripheral blood and bone marrow blasts
Author:
Affiliation:
1. Department of Laboratory Medicine; Cleveland Clinic; Cleveland Ohio
2. Department of Translational Hematology & Oncology Research; Cleveland Clinic; Cleveland Ohio
3. Department of Hematology & Medical Oncology; Cleveland Clinic; Cleveland Ohio
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/ajh.24289/fullpdf
Reference7 articles.
1. inv(16)/t(16;16) acute myeloid leukemia with non-type A CBFB-MYH11 fusions associate with distinct clinical and genetic features and lack KIT mutations;Schwind;Blood,2013
2. Characteristics of translocation (16;16)(p13;q22) acute myeloid leukemia;Eghtedar;Am J Hematol,2012
3. Prognostic factors and outcome of core binding factor acute myeloid leukemia patients with t(8;21) differ from those of patients with inv(16): A Cancer and Leukemia Group B study;Marcucci;J Clin Oncol,2005
4. Pathogenesis of acute myeloid leukaemia and inv(16)(p13;q22): A paradigm for understanding leukaemogenesis?;Reilly;Br J Haematol,2005
5. Core binding factor acute myeloid leukemia;Paschka;Semin Oncol,2008
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