MCPIP1 overexpression in human neuroblastoma cell lines causes cell‐cycle arrest by G1/S checkpoint block-Reference-Cited by-同舟云学术

MCPIP1 overexpression in human neuroblastoma cell lines causes cell‐cycle arrest by G1/S checkpoint block

Published:2020-06 Issue:5-6 Volume:121 Page:3406-3425
ISSN:0730-2312
Container-title:Journal of Cellular Biochemistry
language:en
Short-container-title:J Cell Biochem

Author:

Boratyn Elżbieta¹^ORCID,Nowak Iwona¹^ORCID,Karnas Elżbieta²³^ORCID,Ryszawy Damian³^ORCID,Wnuk Dawid³^ORCID,Polus Anna⁴^ORCID,Durbas Małgorzata¹^ORCID,Horwacik Irena¹^ORCID,Rokita Hanna¹^ORCID

Affiliation:

1. Laboratory of Molecular Genetics and Virology, Faculty of Biochemistry, Biophysics, and BiotechnologyJagiellonian University Kraków Poland

2. Laboratory of Stem Cell Biotechnology, Malopolska Centre of BiotechnologyJagiellonian University Kraków Poland

3. Department of Cell Biology, Faculty of Biochemistry, Biophysics, and BiotechnologyJagiellonian University Kraków Poland

4. Department of Clinical Biochemistry, Medical CollegeJagiellonian University Kraków Poland

Funder

Polish National Science Center

Leading National Research Center

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcb.29614

Reference50 articles.

1. Monocyte chemotactic protein-1-induced protein-1 (MCPIP1) is a novel multifunctional modulator of inflammatory reactions

2. Interleukin-1-inducible MCPIP protein has structural and functional properties of RNase and participates in degradation of IL-1β mRNA

3. MCPIP1 ribonuclease exhibits broad-spectrum antiviral effects through viral RNA binding and degradation

4. Proteasome inhibitor MG ‐132 induces MCPIP 1 expression

5. MCPIP1 Selectively Destabilizes Transcripts Associated with an Antiapoptotic Gene Expression Program in Breast Cancer Cells That Can Elicit Complete Tumor Regression

Cited by 11 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Autophagic degradation of CDK4 is responsible for G0/G1 cell cycle arrest in NVP-BEZ235-treated neuroblastoma;Cancer Biology & Therapy;2024-08

2. Regnase-2 inhibits glioblastoma cell proliferation;Scientific Reports;2024-01-18

3. MCPIP1 functions as a safeguard of early embryonic development;Scientific Reports;2023-10-07

4. Autophagic degradation of CDK4 is responsible for G0/G1 cell cycle arrest in NVP-BEZ235-treated neuroblastoma;2023-05-11

5. The homeostatic function of Regnase‐2 restricts neuroinflammation;The FASEB Journal;2023-02-08