Site selective cyclic amp analogues are antagonistic to estrogen stimulation of growth and proto-oncogene expression in human breast-cancer cells

Author:

Katsaros Dionyssios,Ally Shamsia,Cho-Chung Yoon Sang

Publisher

Wiley

Subject

Cancer Research,Oncology

Reference32 articles.

1. Two classes of cAMP analogs which are selective for the two different cAMP-binding sites of type II protein kinase demonstrate synergism when added together in intact adipocytes;Beebe;J. biol. Chem.,1984

2. Phenol red in tissue culture media is a weak estrogen: implications concerning the study of estrogen-responsive cells in culture;Berthois;Proc. nat. Acad. Sci.,1986

3. Inverse relation between estrogen receptors and cyclic adenosine 3′,5′-monophosphate-binding protein in hormone-dependent mammary tumore regression due to dibutyryl cyclic adenosine 3′.5′-monophosphate treatment or ovariectomy;Bodwin;Cancer Res.,1978

4. The role of cyclic AMP in cell proliferation: a critical assessment of the evidence;Boynton;Advanc. cycl. Nucleotide Res.,1983

5. Hypothesis: cyclic AMP, and its receptor protein in tumor growth regulation in vivo;Cho-Chung;J. cycl. Nucleotide Res.,1980

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