Targeting the TGF-β Receptor with Kinase Inhibitors for Scleroderma Therapy
Author:
Affiliation:
1. Department of Dermatology; General Hospital of Beijing Military Command; Beijing China
2. Graduate School; Third Military Medical University; Chongqing China
Publisher
Wiley
Subject
Drug Discovery,Pharmaceutical Science
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/ardp.201400116/fullpdf
Reference29 articles.
1. Scleroderma
2. Treatment of Scleroderma
3. Getting Out of a Sticky Situation: Targeting the Myofibroblast in Scleroderma
4. Transforming Growth Factor-β Receptor Type 1 (TGFβRI) Kinase Activity but Not p38 Activation Is Required for TGFβRI-Induced Myofibroblast Differentiation and Profibrotic Gene Expression
5. p38 Inhibitors Prevent TGF-β–Induced Myofibroblast Transdifferentiation in Human Tenon Fibroblasts
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