Knockdown of CkrL by shRNA deteriorates hypoxia/reoxygenation-induced H9C2 cardiomyocyte apoptosis and survival inhibition Via Bax and downregulation of P-Erk1/2-Reference-Cited by-同舟云学术

Knockdown of CkrL by shRNA deteriorates hypoxia/reoxygenation-induced H9C2 cardiomyocyte apoptosis and survival inhibition Via Bax and downregulation of P-Erk1/2

Published:2015-02-20 Issue:2 Volume:33 Page:80-88
ISSN:0263-6484
Container-title:Cell Biochemistry and Function
language:en
Short-container-title:Cell Biochem Funct

Author:

Zhang Zhi-Sheng¹²,Yang Dong-Yan¹²,Fu Yan-Bo¹²,Zhang Lei¹²,Zhao Qian-Ping¹,Li Gang¹

Affiliation:

1. Division of Cardiology, Department of Geriatrics; The First Affiliated Hospital of Chongqing Medical University; Chongqing China

2. Laboratory Research Center; The First Affiliated Hospital of Chongqing Medical University; Chongqing China

Funder

the medical science & technology research project of Health Bureau of Chongqing City, P. R. China;Chongqing Science & Technology Commission in Chongqing City, P. R. China

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,General Medicine,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/cbf.3093/fullpdf

Reference33 articles.

1. Cardiomyocyte death: mechanisms and translational implications;Chiong;Cell Death Dis,2011

2. Pro-survival effect of Dock180 overexpression on rat-derived H9C2 cardiomyocytes;Yan;Med Sci Monit Basic Res,2013

3. C3G overexpression promotes the survival of rat-derived H9C2 cardiomyocytes by p-ERK1/2;Zhang;Cell Biol Int,2013

4. Increased expression of Dock180 protein in the noninfarcted myocardium in rats;Liu;J Chin Med Assoc,2013

5. Elevated expression of C3G protein in the peri-infarct myocardium of rats;Wang;Med Sci Monit Basic Res,2013

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