Monocyte‐Derived Interleukin‐1β As the Driver of S100A12‐Induced Sterile Inflammatory Activation of Human Coronary Artery Endothelial Cells: Implications for the Pathogenesis of Kawasaki Disease
Author:
Affiliation:
1. University Children’s Hospital Munster Germany
2. University of Toyama Toyama City Japan
3. University of British ColumbiaBritish Columbia Children’s Hospital Vancouver British Columbia Canada
Funder
Canadian Institutes of Health Research
Michael Smith Foundation for Health Research
Publisher
Wiley
Subject
Immunology,Rheumatology,Immunology and Allergy
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/art.40784
Reference51 articles.
1. IL-1 Inhibition May Have an Important Role in Treating Refractory Kawasaki Disease
2. Diagnosis, Treatment, and Long-Term Management of Kawasaki Disease: A Scientific Statement for Health Professionals From the American Heart Association
3. Neutrophilic involvement in the damage to coronary arteries in acute stage of Kawasaki disease
4. Kawasaki disease: with particular emphasis on arterial lesions;Naoe S;Acta Pathol Jpn,1991
5. Neutrophil-derived S100A12 is profoundly upregulated in the early stage of acute Kawasaki disease
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