Wnt signaling pathway in osteoporosis: Epigenetic regulation, interaction with other signaling pathways, and therapeutic promises
Author:
Affiliation:
1. Department of Genetics Faculty of Medicine, Babol University of Medical Sciences Babol Iran
Publisher
Wiley
Subject
Cell Biology,Clinical Biochemistry,Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.28207
Reference83 articles.
1. Osteoblast-Specific Overexpression of Human WNT16 Increases Both Cortical and Trabecular Bone Mass and Structure in Mice
2. Wnt Proteins Prevent Apoptosis of Both Uncommitted Osteoblast Progenitors and Differentiated Osteoblasts by β-Catenin-dependent and -independent Signaling Cascades Involving Src/ERK and Phosphatidylinositol 3-Kinase/AKT
3. Class I and IIa Histone Deacetylases Have Opposite Effects on Sclerostin Gene Regulation
4. MiR-148a the epigenetic regulator of bone homeostasis is increased in plasma of osteoporotic postmenopausal women
5. Cross-regulation of signaling pathways: An example of nuclear hormone receptors and the canonical Wnt pathway
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