Overexpression of ubiquitin carboxyl terminal hydrolase-L1 enhances multidrug resistance and invasion/metastasis in breast cancer by activating the MAPK/Erk signaling pathway

Author:

Wang Wenjuan1,Zou Liping1,Zhou Danmei1,Zhou Zhongwen1,Tang Feng1,Xu Zude12,Liu Xiuping23

Affiliation:

1. Department of Pathology; Huashan Hospital; Fudan University; Shanghai China

2. Department of Pathology, School of Basic Medical Sciences; Fudan University; Shanghai China

3. Department of Pathology, the Fifth People's Hospital of Shanghai; Fudan University; Shanghai China

Funder

National Nature Science Foundation of China and Shanghai Municipal Commission of Health and Family Planning

Publisher

Wiley

Subject

Cancer Research,Molecular Biology

Reference33 articles.

1. Epigenetic Signatures in Breast Cancer: Clinical Perspective;Parrella;Breast Care (Basel),2010

2. Defining the critical gene expression changes associated with expression and suppression of the tumorigenic and metastatic phenotype in Ha-ras-transformed cloned rat embryo fibroblast cells;Su;Oncogene,1993

3. Involvement of CD147 in regulation of multidrug resistance to P-gp substrate drugs and in vitro invasion in breast cancer cells;Li;Cancer Sci,2004

4. Induction of cancer metastasis by cyclophosphamide pretreatment of host mice: An opposite effect of chemotherapy;Yamauchi;Cancer Res,2008

5. Metastatic potential and multidrug resistance correlation in the B16 melanoma system;Staroselsky;J Exp Ther Oncol,1996

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