Inactivation of the von Hippel–Lindau ( VHL ) tumour suppressor gene and allelic losses at chromosome arm 3p in primary renal cell carcinoma: Evidence for a VHL ‐independent pathway in clear cell renal tumourigenesis-Reference-Cited by-同舟云学术

Inactivation of the von Hippel–Lindau ( VHL ) tumour suppressor gene and allelic losses at chromosome arm 3p in primary renal cell carcinoma: Evidence for a VHL ‐independent pathway in clear cell renal tumourigenesis

Published:1998-07 Issue:3 Volume:22 Page:200-209
ISSN:1045-2257
Container-title:Genes, Chromosomes and Cancer
language:en
Short-container-title:Genes Chromosomes & Cancer

Author:

Clifford Steven C.¹²,Prowse Amanda H.²,Affara Nabeel A.²,Buys Charles H. C. M.³,Maher Eamonn R.¹²

Affiliation:

1. Division of Medical and Molecular Genetics, Department of Paediatrics and Child Health, University of Birmingham, Edgbaston, Birmingham, United Kingdom

2. Department of Pathology, Cambridge University, Cambridge, United Kingdom

3. Department of Medical Genetics, University of Groningen, Groningen, The Netherlands

Publisher

Wiley

Subject

Cancer Research,Genetics

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/%28SICI%291098-2264%28199807%2922%3A3%3C200%3A%3AAID-GCC5%3E3.0.CO%3B2-%23

Reference47 articles.

1. Localization of tumor‐suppressor loci on chromosome‐9 in primary human renal‐cell carcinomas;Cairns P;Cancer Res,1995

2. Epigenetic modification and uniparental inheritance of H19 in Beckwith-Wiedemann syndrome.

3. Suppression of growth of renal carcinoma cells by the von Hippel‐Lindau tumor suppressor gene;Chen F;Cancer Res,1995

4. Identification of intragenic mutations in the Von Hippel — Lindau disease tumour suppressor gene andcorrelation with disease phenotype

5. of heterozygosity at the familial t(3‐8) locus in most clear cell renal carcinomas;Druck T;Cancer Res,1995

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