A protective polymorphism in MMP16 , improved blood gas levels, and chronic obstructive pulmonary diseases: Family and two population‐based studies

Author:

Zhang Zili1ORCID,Wang Jian12,Zheng Zeguang1,Chen Xindong3,Xu Guihua4,Chen Sifan5,Liu Fei1,Chen Lingdan1,Ding Mingjing4,Yuan Liang1,Li Yuanyuan1,Qian Jing1,Xie Xiaohui1,Deng Bingxian1,Lu Wenju12

Affiliation:

1. State Key Laboratory of Respiratory Disease, Guangdong Key Laboratory of Vascular Disease, Guangzhou Institute of Respiratory Health, The First Affiliated Hospital, Guangzhou Medical University Guangdong Guangzhou China

2. Department of Medicine, Division of Translational and Regenerative MedicineThe University of Arizona Tucson Arizona

3. Department of Respiratory MedicineThe First People's Hospital of Lufeng Lufeng Guangdong China

4. Department of Respiratory MedicineInner Mongolia Autonomous Region People's Hospital Hohhot Inner Mongolia Autonomous Region China

5. Guangdong Provincial Key Laboratory of Malignant Tumor Epigenetics and Gene Regulation, Medical Research Center, Sun Yat‐Sen Memorial Hospital, Sun Yat‐Sen University Guangzhou China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Genetics (clinical),Genetics

Reference40 articles.

1. Pathogenesis of COPD. Part I. The role of protease‐antiprotease imbalance in emphysema;Abboud R. T.;International Journal of Tuberculosis and Lung Disease,2008

2. A new look at the statistical model identification

3. Membrane type‐1 matrix metalloproteinase (MT1‐MMP) correlates with the expression and activation of matrix metalloproteinase‐2 (MMP‐2) in inflammatory breast cancer;Al‐Raawi D.;International Journal of Clinical and Experimental Medicine,2011

4. Association of MMP-2 Activation Potential With Metastatic Progression in Human Breast Cancer Cell Lines Independent of MMP-2 Production

5. Roles of microRNA-206 in Osteosarcoma Pathogenesis and Progression

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