Concurrent STAT3, DNMT3A, and TET2 mutations in T-LGL leukemia with molecularly distinct clonal hematopoiesis of indeterminate potential
Author:
Affiliation:
1. Department of Pathology; Oregon Health and Science University; Portland Oregon
2. Knight Cancer Institute, Oregon Health and Science University; Portland Oregon
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/ajh.24586/fullpdf
Reference15 articles.
1. Somatic STAT3 mutations in large granular lymphocytic leukemia;Koskela;N Engl J Med,2012
2. Frequent STAT5B mutations in gammadelta hepatosplenic T-cell lymphomas;Nicolae;Leukemia,2014
3. STAT3 mutations are frequent in CD30+ T-cell lymphomas and T-cell large granular lymphocytic leukemia;Ohgami;Leukemia,2013
4. Activating mutations of STAT5B and STAT3 in lymphomas derived from gammadelta-T or NK cells;Kucuk;Nat Commun,2015
5. Mutations in the JAK/STAT and RAS signaling pathways are common in intestinal T-cell lymphomas;Nicolae;Leukemia,2016
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