Tumor Cell‐Intrinsic SETD2 Deficiency Reprograms Neutrophils to Foster Immune Escape in Pancreatic Tumorigenesis-Reference-Cited by-全球学者库

Tumor Cell‐Intrinsic SETD2 Deficiency Reprograms Neutrophils to Foster Immune Escape in Pancreatic Tumorigenesis

Published:2022-12 Issue:2 Volume:10 Page:2202937
ISSN:2198-3844
Container-title:Advanced Science
language:en
Short-container-title:Advanced Science

Author:

Niu Ningning¹,Shen Xuqing¹,Zhang Li¹,Chen Yueyue¹,Lu Ping¹,Yang Wenjuan¹,Liu Mingzhu¹,Shi Juanjuan¹,Xu Dapeng²,Tang Yingying¹,Yang Xiaotong¹,Weng Yawen¹,Zhao Xinxin³,Wu Lian‐Ming³,Sun Yongwei²,Xue Jing¹^ORCID

Affiliation:

1. State Key Laboratory of Oncogenes and Related Genes Stem Cell Research Center Shanghai Cancer Institute Ren Ji Hospital Shanghai Jiao Tong University School of Medicine 160 Pujian Rd Shanghai 200127 P. R. China

2. Department of Biliary‐Pancreatic Surgery Ren Ji Hospital Shanghai Jiao Tong University School of Medicine 160 Pujian Rd Shanghai 200127 P. R. China

3. Department of Radiology Ren Ji Hospital Shanghai Jiao Tong University School of Medicine 160 Pujian Rd Shanghai 200127 P. R. China

Funder

National Natural Science Foundation of China

Science and Technology Commission of Shanghai Municipality

Publisher

Wiley

Subject

General Physics and Astronomy,General Engineering,Biochemistry, Genetics and Molecular Biology (miscellaneous),General Materials Science,General Chemical Engineering,Medicine (miscellaneous)

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/advs.202202937

Reference42 articles.

1. Immune crosstalk in cancer progression and metastatic spread: a complex conversation

2. Tumour-intrinsic resistance to immune checkpoint blockade

3. The Tumor Microenvironment Innately Modulates Cancer Progression

4. Mutant KRAS–Induced Expression of ICAM-1 in Pancreatic Acinar Cells Causes Attraction of Macrophages to Expedite the Formation of Precancerous Lesions

5. In Vivo Epigenetic CRISPR Screen Identifies Asf1a as an Immunotherapeutic Target in Kras-Mutant Lung Adenocarcinoma

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1. The Role of Chemokines in Orchestrating the Immune Response to Pancreatic Ductal Adenocarcinoma;Cancers;2024-01-28

2. The role of innate immune cells as modulators of the tumor microenvironment in the metastasis and treatment of pancreatic cancer;Clinical Cancer Bulletin;2023-12-06

3. Tumor cell-intrinsic SETD2 inactivation sensitizes cancer cells to immune checkpoint blockade through the NR2F1-STAT1 pathway;Journal for ImmunoTherapy of Cancer;2023-12

4. Tumor-suppressive functions of protein lysine methyltransferases;Experimental & Molecular Medicine;2023-12-01

5. Histone methyltransferase SETD2 inhibits M1 macrophage polarization and glycolysis by suppressing HIF-1α in sepsis-induced acute lung injury;Medical Microbiology and Immunology;2023-09-01