The combined inhibition of the CaMKIIδ and calcineurin signaling cascade attenuates IGF‐IIR‐induced cardiac hypertrophy-Reference-Cited by-同舟云学术

The combined inhibition of the CaMKIIδ and calcineurin signaling cascade attenuates IGF‐IIR‐induced cardiac hypertrophy

Published:2019-10-04 Issue:4 Volume:235 Page:3539-3547
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:J Cell Physiol

Author:

Chen Chung‐Hao¹,Lin Jing‐Wei²,Huang Chih‐Yang³^ORCID,Yeh Yu‐Lan⁴,Shen Chia‐Yao⁵,Badrealam Khan Farheen⁶^ORCID,Ho Tsung‐Jung⁷,Padma V. Vijaya⁸,Kuo Wei‐Wen⁹^ORCID,Huang Chih‐Yang¹⁰⁶¹¹¹²¹³^ORCID

Affiliation:

1. Graduate Institute of Aging Medicine China Medical University Taichung Taiwan

2. Graduate Institute of Biomedical Science China Medical University Taichung Taiwan

3. Translation Research Core China Medical University Hospital Taichung Taiwan

4. Department of Pathology Changhua Christian Hospital Changhua Taiwan

5. Department of Nursing Mei Ho University Pingtung Taiwan

6. Cardiovascular and Mitochondria Related Diseases Research Center, Hualien Tzu Chi Hospital Buddhist Tzu Chi Medical Foundation Hualien Taiwan

7. Department of Chinese Medicine, Hualien Tzu Chi Hospital, Buddhist Tzu Chi Medical Foundation Tzu Chi University Hualien Taiwan

8. Department of Biotechnology Bharathiar University Coimbatore India

9. Department of Biological Science and Technology China Medical University Taichung Taiwan

10. Graduate Institute of Biomedical Sciences China Medical University Taichung Taiwan

11. Center of General Education, Buddhist Tzu Chi Medical Foundation Tzu Chi University of Science and Technology Hualien Taiwan

12. Department of Medical Research, China Medical Hospital China Medical University Taichung Taiwan

13. Department of Biotechnology Asia University Taichung Taiwan

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.29242

Reference26 articles.

1. ECM remodeling in hypertensive heart disease

2. [Leu27] INSULIN-LIKE GROWTH FACTOR II IS HIGHLY SELECTIVE FOR THE TYPE-II IGF RECEPTOR IN BINDING, CROSS-LINKING AND THYMIDINE INCORPORATION EXPERIMENTS

3. IGF-II/mannose 6-phosphate receptor activation induces metalloproteinase-9 matrix activity and increases plasminogen activator expression in H9c2 cardiomyoblast cells

4. Tanshinone IIA Inhibits β-Catenin Nuclear Translocation and IGF-2R Activation via Estrogen Receptors to Suppress Angiotensin II-Induced H9c2 Cardiomyoblast Cell Apoptosis

5. IGF-II/mannose-6-phosphate receptor signaling induced cell hypertrophy and atrial natriuretic peptide/BNP expression via Gαq interaction and protein kinase C-α/CaMKII activation in H9c2 cardiomyoblast cells

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