Antiglycation potential of plant based TiO <sub>2</sub> nanoparticle in D‐ribose glycated BSA in vitro-Reference-Cited by-同舟云学术

Antiglycation potential of plant based TiO ₂ nanoparticle in D‐ribose glycated BSA in vitro

Published:2022-09-21 Issue:7 Volume:40 Page:784-796
ISSN:0263-6484
Container-title:Cell Biochemistry and Function
language:en
Short-container-title:Cell Biochemistry & Function

Author:

Alenazi Fahaad¹,Saleem Mohd²,Syed Khaja Azharuddin Sajid²^ORCID,Zafar Mubashir³,Alharbi Mohammed Salem⁴,Al Hagbani Turki⁵^ORCID,Khan Mohd Yasir⁶^ORCID,Ahmad Waseem⁷,Ahmad Saheem⁸^ORCID

Affiliation:

1. Department of Pharmacology, College of Medicine University of Hail Hail Saudi Arabia

2. Department of Pathology, College of Medicine University of Hail Hail Saudi Arabia

3. Department of Community Medicine, College of Medicine University of Hail Hail Saudi Arabia

4. Department of Internal Medicine, College of Medicine University of Hail Hail Saudi Arabia

5. Department of Pharmaceutics, College of Pharmacy University of Hail Hail Saudi Arabia

6. Department of Biotechnology, School of Applied & Life Science (SALS) Uttaranchal University Dehradun Uttarakhand India

7. Department of Chemistry, School of Applied & Life Science (SALS) Uttaranchal University Dehradun Uttarakhand India

8. Department of Medical Laboratory Sciences, College of Applied Medical Sciences University of Hail Hail Saudi Arabia

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,General Medicine,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/cbf.3744

Reference58 articles.

1. Endothelial Dysfunction in Diabetes Is Aggravated by Glycated Lipoproteins; Novel Molecular Therapies

2. The Effect of Intensive Glycemic Treatment on Coronary Artery Calcification in Type 1 Diabetic Participants of the Diabetes Control and Complications Trial/Epidemiology of Diabetes Interventions and Complications (DCCT/EDIC) Study

3. Intensive Diabetes Treatment and Cardiovascular Disease in Patients with Type 1 Diabetes

4. Metformin attenuates fluctuating glucose-induced endothelial dysfunction through enhancing GTPCH1-mediated eNOS recoupling and inhibiting NADPH oxidase

5. Changes in Diabetes-Related Complications in the United States, 1990–2010

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