SUMOylated αNAC Potentiates Transcriptional Repression by FIAT
Author:
Affiliation:
1. Research Unit; Shriners Hospitals for Children - Canada; Montreal Quebec Canada H3G 1A6
2. Department of Human Genetics; McGill University; Montreal Quebec Canada H3A 1B1
Publisher
Wiley
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference34 articles.
1. Sequence-specific DNA binding by the alphaNAC coactivator is required for potentiation of c-Jun-dependent transcription of the Osteocalcin gene;Akhouayri;Mol Cell Biol,2005
2. Inhibition of parathyroid hormone responsiveness in clonal osteoblastic cells expressing a mutant form of 3',5'-cyclic adenosine monophosphate-dependent protein kinase;Bringhurst;Mol Endocrinol,1989
3. Accurate transcription initiation by RNA polymerase II in a soluble extract from isolated mammalian nuclei;Dignam;Nucleic Acids Res,1983
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1. Dephosphorylation of the transcriptional cofactor NACA by the PP1A phosphatase enhances cJUN transcriptional activity and osteoblast differentiation;Journal of Biological Chemistry;2019-05
2. Lrp6 is a target of the PTH-activated αNAC transcriptional coregulator;Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms;2018-02
3. New PTH Signals Mediating Bone Anabolism;Current Molecular Biology Reports;2017-04-22
4. Altered gene dosage confirms the genetic interaction between FIAT and αNAC;Gene;2014-04
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