A requirement for septins and the autophagy receptor p62 in the proliferation of intracellular Shigella-Reference-Cited by-同舟云学术

A requirement for septins and the autophagy receptor p62 in the proliferation of intracellular Shigella

Published:2018-09-10 Issue:1 Volume:76 Page:163-172
ISSN:1949-3584
Container-title:Cytoskeleton
language:en
Short-container-title:Cytoskeleton

Author:

Lobato‐Márquez Damián¹²^ORCID,Krokowski Sina¹²,Sirianni Andrea¹,Larrouy‐Maumus Gerald³,Mostowy Serge¹²^ORCID

Affiliation:

1. MRC Centre for Molecular Bacteriology and Infection, Department of MedicineSection of Microbiology, Imperial College London London United Kingdom

2. Department of Immunology and InfectionLondon School of Hygiene and Tropical Medicine, Keppel Street London United Kingdom

3. MRC Centre for Molecular Bacteriology and Infection, Department of Life Sciences, Faculty of Natural SciencesImperial College London London United Kingdom

Funder

Lister Institute of Preventive Medicine

Publisher

Wiley

Subject

Cell Biology,Structural Biology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/cm.21453

Reference57 articles.

1. Host Delivery of Favorite Meals for Intracellular Pathogens

2. A glycolytic metabolon inSaccharomyces cerevisiaeis stabilized by F-actin

3. Septins are involved at the early stages of macroautophagy in S. cerevisiae

4. The Legionella Effector RavZ Inhibits Host Autophagy Through Irreversible Atg8 Deconjugation

5. Out of Asia: The independent rise and global spread of fluoroquinolone‐resistant Shigella;Chung H.;Microbial Genomics,2018

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5. TARC and Septin 7 can be better monitoring biomarkers than CX3CL1, sICAM5, and IRF5 in children with seizure-free epilepsy with monotherapy and drug-resistant epilepsy;International Journal of Neuroscience;2022-08-10