Quantitative proteomic study of myocardial mitochondria in urea transporter B knockout mice

Author:

Du Yanwei1,Meng Yan1,Zhu Jun2,Kang Le1,Jia Xiaolong1,Guo Lirong1,Zhang Ling1,Ye Mingliang2,Hu Lianghai3,Zhao Xuejian1,Gu Jingkai3,Yang Baoxue14,Zou Hanfa2

Affiliation:

1. Key Laboratory of Pathobiology; Department of Pathophysiology; Ministry of Education; College of Basic Medicine; Jilin University; Changchun P. R. China

2. CAS Key Laboratory of Separation Science for Analytical Chemistry; Dalian Institute of Chemical Physics; Chinese Academy of Sciences; Dalian P. R. China

3. Key Laboratory for Molecular Enzymology & Engineering; Ministry of Education; School of Life Sciences; Jilin University; Changchun P. R. China

4. State Key Laboratory of Natural and Biomimetic Drugs; Key Laboratory of Molecular Cardiovascular Sciences; Ministry of Education; Department of Pharmacology; School of Basic Medical Sciences; Peking University; Beijing P. R. China

Funder

National Natural Science Foundation of China

New Century Excellent Talents in University

Publisher

Wiley

Subject

Molecular Biology,Biochemistry

Reference39 articles.

1. Urinary concentrating ability in patients with Jk(a-b-) blood type who lack carrier-mediated urea transport;Sands;J. Am. Soc. Nephrol.,1992

2. Urea and urine concentrating ability: new insights from studies in mice;Yang;Am. J. Physiol. Renal Physiol.,2005

3. Molecular mechanisms of urea transport;Sands;J. Membr. Biol.,2003

4. Kidd blood group and urea transport function of human erythrocytes are carried by the same protein;Olives;J. Biol. Chem.,1995

5. A novel mutation at the JK locus causing Jk null phenotype in a Chinese family;Meng;Sci. China C Life Sci.,2005

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