EZH2 Supports Osteoclast Differentiation and Bone Resorption Via Epigenetic and Cytoplasmic Targets-Reference-Cited by-同舟云学术

EZH2 Supports Osteoclast Differentiation and Bone Resorption Via Epigenetic and Cytoplasmic Targets

Published:2019-10-23 Issue:1 Volume:35 Page:181-195
ISSN:0884-0431
Container-title:Journal of Bone and Mineral Research
language:en
Short-container-title:J Bone Miner Res

Author:

Adamik Juraj¹^ORCID,Pulugulla Sree H²^ORCID,Zhang Peng¹,Sun Quanhong¹,Lontos Konstantinos¹,Macar David A²,Auron Philip E²^ORCID,Galson Deborah L¹³^ORCID

Affiliation:

1. Department of Medicine, Division of Hematology‐Oncology, UPMC Hillman Cancer Center University of Pittsburgh School of Medicine Pittsburgh PA USA

2. Department of Biological Sciences Duquesne University Pittsburgh PA USA

3. McGowan Institute for Regenerative Medicine University of Pittsburgh Pittsburgh PA USA

Funder

Duquesne University

National Cancer Institute

National Institute of Arthritis and Musculoskeletal and Skin Diseases

National Science Foundation

Publisher

Wiley

Subject

Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jbmr.3863

Reference63 articles.

1. PU.1 target genes undergo Tet2-coupled demethylation and DNMT3b-mediated methylation in monocyte-to-osteoclast differentiation

2. Identification of Novel Transcription Factors in Osteoclast Differentiation Using Genome-wide Analysis of Open Chromatin Determined by DNase-seq

3. Inhibition of the histone demethylase KDM4B leads to activation of KDM1A, attenuates bacterial-induced pro-inflammatory cytokine release, and reduces osteoclastogenesis

4. DNA methyltransferase 3a regulates osteoclast differentiation by coupling to an S-adenosylmethionine–producing metabolic pathway

5. Trichostatin A inhibits osteoclastogenesis and bone resorption by suppressing the induction of c-Fos by RANKL

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