CD55 Regulates Bone Mass in Mice by Modulating RANKL‐Mediated Rac Signaling and Osteoclast Function-Reference-Cited by-同舟云学术

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CD55 Regulates Bone Mass in Mice by Modulating RANKL‐Mediated Rac Signaling and Osteoclast Function

Published:2019-10-23 Issue:1 Volume:35 Page:130-142
ISSN:0884-0431
Container-title:Journal of Bone and Mineral Research
language:en
Short-container-title:J Bone Miner Res

Author:

Shin Bongjin¹,Won Heeyeon¹,Adams Douglas J²³,Lee Sun‐Kyeong¹^ORCID

Affiliation:

1. UCONN Center on AgingUniversity of Connecticut Health Center Farmington CT USA

2. Department of Orthopedic SurgeryUniversity of Connecticut Health Center Farmington CT USA

3. Department of OrthopedicsUniversity of Colorado, Anschutz Medical Campus Aurora CO USA

Publisher

Wiley

Subject

Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jbmr.3861

Reference63 articles.

1. Genetic regulation of osteoclast development and function

2. Osteoimmunology: Interactions of the Bone and Immune System

3. Osteoimmunology: cytokines and the skeletal system

4. alphavbeta3 and macrophage colony-stimulating factor: partners in osteoclast biology

5. Osteoclast precursors, RANKL/RANK, and immunology

Cited by 10 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Cellular taxonomy of the preleukemic bone marrow niche of acute myeloid leukemia;2023-09-04

2. Physiological functions of podosomes: From structure and function to therapy implications in osteoclast biology of bone resorption;Ageing Research Reviews;2023-03

3. CD55 in cancer: Complementing functions in a non-canonical manner;Cancer Letters;2022-12

4. CD55 is upregulated by cAMP/PKA/AKT and modulates human decidualization via Src and ERK pathway and decidualization‐related genes;Molecular Reproduction and Development;2022-04-26

5. Inhibition of ACLY Leads to Suppression of Osteoclast Differentiation and Function Via Regulation of Histone Acetylation;Journal of Bone and Mineral Research;2021-07-26

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