Heat-shock protein-90 prolongs septic neutrophil survival by protecting c-Src kinase and caspase-8 from proteasomal degradation-Reference-Cited by-同舟云学术

Heat-shock protein-90 prolongs septic neutrophil survival by protecting c-Src kinase and caspase-8 from proteasomal degradation

Published:2018-02-02 Issue:5 Volume:103 Page:933-944
ISSN:0741-5400
Container-title:Journal of Leukocyte Biology
language:en
Short-container-title:J Leukoc Biol

Author:

Gupta Sahil¹²³,Lee Chan-Mi⁴⁵,Wang Jia-Feng⁶,Parodo Jean¹²,Jia Song-Hui¹²,Hu Jim⁴⁵,Marshall John C.¹²³

Affiliation:

1. The Keenan Research Centre for Biomedical Science; Li Ka Shing Knowledge Institute, St. Michael's Hospital; Toronto Ontario Canada

2. Departments of Surgery and Critical Care Medicine; St. Michael's Hospital, University of Toronto; Toronto Ontario Canada

3. Institute of Medical Science, Faculty of Medicine; University of Toronto; Toronto Ontario Canada

4. Department of Laboratory Medicine and Pathobiology; University of Toronto; Toronto Ontario Canada

5. Hospital for Sick Children Research Institute, Program in Physiology and Experimental Medicine, Peter Gilgan Centre for Research and Learning, Hospital for Sick Children; Toronto Ontario Canada

6. Department of Anesthesiology and Intensive Care; Changhai Hospital, The Second Military Medical University; Shanghai China

Funder

Canadian Institutes of Health Research

Physicians' Services Incorporated Foundation

Publisher

Wiley

Subject

Cell Biology,Immunology,Immunology and Allergy

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4. Inhibitory effects of heat shock protein 90 blockade on proinflammatory human Th1 and Th17 cell subpopulations;Tukaj;J Inflamm (Lond),2014

5. Why have clinical trials in sepsis failed?;Marshall;Trends Mol Med,2014

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