Tumour necrosis factor-α drives Alport glomerulosclerosis in mice by promoting podocyte apoptosis-Reference-Cited by-同舟云学术

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Tumour necrosis factor-α drives Alport glomerulosclerosis in mice by promoting podocyte apoptosis

Published:2011-09-26 Issue:1 Volume:226 Page:120-131
ISSN:0022-3417
Container-title:The Journal of Pathology
language:en
Short-container-title:J. Pathol.

Author:

Ryu Mi,Mulay Shrikant R,Miosge Nicolai,Gross Oliver,Anders Hans-Joachim

Funder

Association pour l'Information et la Recherche sur les Maladies Rénale Génétiques, France

Publisher

Wiley

Subject

Pathology and Forensic Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/path.2979/fullpdf

Reference35 articles.

1. Type IV collagen: structure, gene organization, and role in human diseases. Molecular basis of Goodpasture and Alport syndromes and diffuse leiomyomatosis;Hudson;J Biol Chem,1993

2. Stage-specific action of matrix metalloproteinases influences progressive hereditary kidney disease;Zeisberg;PLoS Med,2006

3. Integrin alpha1beta1 and transforming growth factor-beta1 play distinct roles in Alport glomerular pathogenesis and serve as dual targets for metabolic therapy;Cosgrove;Am J Pathol,2000

4. The renal lesions of Alport syndrome;Heidet;J Am Soc Nephrol,2009

5. Alport's syndrome, Goodpasture's syndrome, and type IV collagen;Hudson;N Engl J Med,2003

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2. GLP-1 receptor agonist, liraglutide, protects podocytes from apoptosis in diabetic nephropathy by promoting white fat browning;Biochemical and Biophysical Research Communications;2023-07

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4. TNF-α-mediated podocyte injury via the apoptotic death receptor pathway in a mouse model of IgA nephropathy;Renal Failure;2022-07-14

5. Urinary Monocyte Chemoattractant Protein-1 in Patients With Alport Syndrome;Kidney International Reports;2022-05

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